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Brain-focused conclusions require an appropriate combination of neural tissue, cerebrospinal fluid, imaging, or mechanistic models

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Together, these studies argue that FSP1 can represent (1) a context-dependent essentiality (for example, metastatic niches) and/or (2) a stand-alone oxidative defense node that becomes rate limiting for tumor survival under physiological stressors encountered in vivo

Co-expression with COP1 substrates including c-Jun or ETS2 disrupts this configuration, inducing a conformational rearrangement into a distinct dimeric state that allows substrate access

[3] These systems are central to mood regulation, motivation, and executive cognitive function